I wrote a detailed answer to this but clicked an old bookmark and it's gone.
If you have genuinely looked, then you haven't understood what you've read. Do a search on "How natural covid antibodies differ from"
Perhaps you remain too uneducated to undeerstand much, certainly to say much.
You certainly haven't read what I wrote before.
You take the píss out of what you don't understand.
Some of what you're saying is plain ignorant, really not worth anyone's time, sorry.
One more go:
You need to learn about what's in the virus, and which bits the western vaccines target, and what the crude treatments which produce "inactivated" vaccines , like heat, pH, UV or detergents, (in the case of lipid coated ones like covid), etc, often do. Inactivated viruses do not provide the same antibodies as the original virus. These vaccines aree not synthesised codon at a time.
There is some bad hsitory.. You need to know something of the other problems associated with traditional inactivated vaccines which will tell you why the west doesn't like them.
If you're unlucky you produce a more vaccine-resistant strain in the environment. They take a long time to develop and
test, to be effective and safe.
That's a main reason why we in the west are not using them.
Despite what some say, vaccines usually get more testing that the ones we're using, have had time for.
However much you like to champion eg SInopharm, it's not great in some tests
https://www.jpost.com/health-and-we...-v-more-effective-than-sinopharm-study-679130 https://en.wikipedia.org/wiki/Sinopharm_BIBP_COVID-19_vaccine though it depends what you measure. Beware Chinese testers.
You will know that we (Pf, M, Az, & others ....) are
only spike-targeting vaccines. The S (spike) potein collection iis how the virsu "gets in". It alters, so the western virus tried to cover variations. Some fail rather, eg AstaZenica and the beta variant.
LEarn how memory antibodies (T and B) evolve to broaden their effect and it weakens over time.
Our covid molecule has four major structural proteins (E, M, N and S) and 16 non-structural proteins encoded by ORF1a and ORF1b together (ORF1ab) that are involved in virus pathogenicity and infectivity*. There's a lot of work going on to try to sort out what deas what, to us. We know some about some problems, eg S and blood.
There are at least dozens of epitopes, 50 odd in the S , yes? Loads more inthe rest...
You need to understand something of the etiology and pathogenicity of virus-contained proteins. I bet you don't know much about that because nobody else has it all. We know fairly well what they do for the the virus, but not all about how they damage us. We're still working on eg how the loss of taste and smell works, and how exactly the cytokine system is disrupted.
Many of the syptoms of long covid, as with a number of other chronic conditions, we don't know much about. May be neuro damage.
Over time vaccines will evolve to cover some of those, but we aren't there yet.
The variants to some extent still manage to bind with ACE-2 you should know about, or elsewhere or get in somehow, possibly via ways we don't have cracked yet - research still ongoing on that.
That means that quadruple jabbing with the same vaccines probably won't stop them all - we need more approaches.^#
Here's a few links. SUggest you read a couple of dozen more from different sources, to get the general idea.
*
https://pubmed.ncbi.nlm.nih.gov/34118359/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7138183/
https://www.miragenews.com/natural-infection-versus-vaccination-619201/
https://www.nih.gov/news-events/nih-research-matters/how-covid-19-variants-evade-immune-response
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4147684/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7189890/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7488171/
https://pubmed.ncbi.nlm.nih.gov/32645228/ if you're fat
^
https://scitechdaily.com/decoy-rece...-cov-2-covid-19-coronavirus-in-cell-cultures/
#
https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(20)32137-1/fulltext
